Jugular Venous Pressure

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Jugular venous pressure (JVP) provides an indirect measure of central venous pressure. The internal jugular vein connects to the right atrium without any intervening valves - thus acting as a column for the blood in the right atrium. The JVP consists of certain waveforms and abnormalities of these can help to diagnose certain conditions.[1] Unfortunately, detection of these abnormalities and even the JVP itself, can be difficult and has also been superseded by other diagnostic methods.

  • Use the right internal jugular vein (IJV).
  • The patient should be at a 45° angle.
  • The patient's head should be turned slightly to the left.
  • If possible, have a tangential light source that shines obliquely from the left.
  • Locate the surface markings of the IJV - this runs from the medial end of the clavicle to the ear lobe, under the medial aspect of the sternocleidomastoid.
  • Locate the JVP - look for the double waveform pulsation (palpating the contralateral carotid pulse will help).
  • Measure the level of the JVP by measuring the vertical distance between the sternal angle and the top of the JVP. Measure the height - usually less than 3 cm.

For a diagram, see 'JVP Waveforms' under 'Document references', below.[5]

Waves[2][3][4]
  • a - presystolic; produced by right atrial contraction.
  • c - bulging of the tricuspid valve into the right atrium during ventricular systole (isovolumic phase).
  • v - occurs in late systole; increased blood in the right atrium from venous return.
Descents
  • x - a combination of atrial relaxation, downward movement of the tricuspid valve and ventricular systole.
  • y - the tricuspid valve opens and blood flows into the right ventricle.

The a and v waves can be identified by timing the double waveform with the opposite carotid pulse. The a wave will occur just before the pulse and the v wave occurs towards the end of the pulse. Distinguishing the c wave, x and y descents is an almost impossible task.

The jugular venous pulse is:
  • Not palpable.
  • Obliterated by pressure.
  • Characterised by a double waveform.
  • Variable with respiration - it decreases with inspiration.
  • Enhanced by the hepatojugular reflux (see below).
  • This can help to confirm that the pulsation is caused by the JVP.
  • Firm pressure is applied to the right upper quadrant using the palm of the hand.
  • A transient increase in the JVP will be seen in normal patients.
  • There may be a delayed recovery back to baseline which is more marked in right ventricular failure.

Abnormalities of the a wave

Prominent v waves

  • Tricuspid regurgitation - called cv or v waves and occurring at the same time as systole (a combination of v wave and loss of x descent); there may be earlobe movement.

Slow y descent

Steep y descent

  • Right ventricular failure.
  • Constrictive pericarditis.
  • Tricuspid regurgitation.
  • (The last two conditions have a rapid rise and fall of the JVP - called Friedreich's sign.)

Elevated JVP in patients with heart failure is associated with an increased risk of hospital admission, death and subsequent hospitalisation for heart failure.[7] Therefore, appreciation of this sign can be clinically helpful.

Further reading & references

  1. Jevon P, Cunnington A; Cardiovascular examination. Part one of a four-part series. Measuring jugular venous pressure. Nurs Times. 2007 Jun 19-25;103(25):28-9.
  2. Kumar P, Clarke M; Clinical Medicine, 6th Ed, (2005). WB Saunders: London
  3. Harrison's Principles of Internal Medicine, 15th Ed. Eds: Braunwald, E et al. McGraw-Hill, USA, 2001
  4. Souhami, RL and Moxham, J (Eds). Textbook of medicine, 4th edition, (2002), Churchill Livingstone: China
  5. JVP Waveforms, University of California - Hospitalist Handbook, 2002; provides a good image of jugular venous pulsations
  6. Wiese J; The abdominojugular reflux sign. Am J Med. 2000 Jul;109(1):59-61.
  7. Drazner MH, Rame JE, Stevenson LW, et al; Prognostic importance of elevated jugular venous pressure and a third heart sound in patients with heart failure. N Engl J Med. 2001 Aug 23;345(8):574-81.

Disclaimer: This article is for information only and should not be used for the diagnosis or treatment of medical conditions. EMIS has used all reasonable care in compiling the information but make no warranty as to its accuracy. Consult a doctor or other health care professional for diagnosis and treatment of medical conditions. For details see our conditions.

Original Author:
Dr Gurvinder Rull
Current Version:
Peer Reviewer:
Dr Hannah Gronow
Last Checked:
28/09/2011
Document ID:
2350 (v23)
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